Breast Cancer can Change if it Spreads to Other Parts of the Body
November 4, 2009
Boston (DbTechNo) - Results of a new study show that many breast cancers actually change as they spread to other parts of the human body.
The study, published in the Annals of Oncology shows that up to 40% of breast cancers can change form when they spread, a fact that could result in the differing of treatment to help combat the disease.
Researchers analyzed 211 breast cancer tumors, which had spread to the lymph nodes of patients.
They noted that when the cancer cells had migrated into the lymph nodes of patients, the disease became much more difficult to treat.
Lead researcher Dr Dana Faratian said: “We were surprised that such a high proportion of tumours change form when they spread beyond the breast.
“This suggests there is a need to test which type of disease a woman has in the lymph nodes, because it could radically alter the course of treatment she receives.
In the UK alone, it is estimated that 1 in 9 women will be diagnosed with breast cancer during their lifetime.
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Prostaglandins are infinitesimal, ephemeral, and powerful signaling molecules that self-regulate the chemistry of every cell in the body, including cells regulating mood, and those regulating immune function. When produced within normal limits, prostaglandins regulate the physiology of every cell; when produced excessively, physiology becomes pathology. When brain cells produce excessive concentrations of prostaglandins, they depress mood and immunity. In 1973, David Horrobin showed that antidepressants inhibit prostaglandins, and in 1977 that prostaglandins regulate nucleic acids (DNA and RNA).1,2 Others subsequently showed that prostaglandins regulate the synthesis, inhibition, and expression of genes, and the growth and replication of cells, with cancer the accelerated replication of abnormal cells.1,2 Excessive synthesis of prostaglandins induces cancer, with genes determining the variations. In 1998 Brenda Penninx published a key study showing that age 70 chronically depressed people have an increased risk of 88% of developing cancer, and of 50% of dying of it.
More than sixty studies have shown that antidepressants kill cancer cells, inhibit their proliferation, convert multidrug resistant cells to sensitive, protect nonmalignant cells from damage by radiation and chemotherapy toxicity, and target the mitochondria of cancer cells while sparing those of healthy ones.1,2 Antidepressants have therapeutic potential in many cancers that are often treatment resistant, such as gliomas, cancers of the lung, kidney, liver, and uterus, inflammatory breast cancer, and multiple myelomas.2 Antidepressants are capable of arresting cancer in advanced stages, and even reversing it. That antidepressants are effective for a multitude of malignancies, decries the myth that cancer is a hundred diseases, when it is one disease with a hundred variations. Antidepressants alleviate cancer pain, alone or combined with narcotics, remit nausea and vomiting, promote sleep, relieve anxiety and depression, and combat fatigue. Other inhibitors of prostaglandins, such as non-steroidal, anti-inflammatory drugs as aspirin, indomethacin, ibuprofen, and piroxicam, and COX-2 inhibitors, also have potential value in defeating cancer.
Sincerely,
Julian Lieb, M.D
1.Lieb, J.”The multifaceted value of antidepressants in cancer therapeutics.” Eur J Cancer. Editorial Comment. (2008) 172-174
2.Lieb, J.”Defeating cancer with antidepressants.” ecancermedicalscience DOI.10.3332/eCMS.2008.88
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